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An Overview of the Molecular Mechanisms Associated with Myocardial Ischemic Injury: State of the Art and Translational Perspectives

by Valenti, Valentina , Sarto, Gianmarco , Petrozza, Vincenzo , Schiavon, Sonia , Spinosa, Giulia , Forte, Maurizio , Sciarretta, Sebastiano , Vecchio, Daniele , Frati, Giacomo , D’Ambrosio, Luca , Schirone, Leonardo

in Adenosine triphosphate / Autophagy / Cardiomyocytes / Cardiovascular disease / Cardiovascular diseases / Congestive heart failure / Coronary vessels / Diabetes / Heart / Heart attacks / Heart Injuries / Humans / infarction / Inflammation / Ischemia / ischemia/reperfusion (I/R) / ischemic injury / Kinases / mechanisms / Metabolism / Mitochondria / molecular / Molecular modelling / Myocardial infarction / Myocardial Infarction - metabolism / myocardial ischemia / Myocardial Reperfusion Injury - metabolism / Nitric oxide / Oxidative stress / Phosphatase / Phosphorylation / Reperfusion / Review / Smooth muscle / Therapeutic applications / Veins & arteries / Ventricular Remodeling

2022

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An Overview of the Molecular Mechanisms Associated with Myocardial Ischemic Injury: State of the Art and Translational Perspectives

2022

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An Overview of the Molecular Mechanisms Associated with Myocardial Ischemic Injury: State of the Art and Translational Perspectives

2022

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Journal Article

An Overview of the Molecular Mechanisms Associated with Myocardial Ischemic Injury: State of the Art and Translational Perspectives

Valenti, Valentina,

Sarto, Gianmarco,

Petrozza, Vincenzo,

Schiavon, Sonia,

Spinosa, Giulia,

Forte, Maurizio,

Sciarretta, Sebastiano,

Vecchio, Daniele,

Frati, Giacomo,

D’Ambrosio, Luca,

Schirone, Leonardo

2022

Overview

Cardiovascular disease is the leading cause of death in western countries. Among cardiovascular diseases, myocardial infarction represents a life-threatening condition predisposing to the development of heart failure. In recent decades, much effort has been invested in studying the molecular mechanisms underlying the development and progression of ischemia/reperfusion (I/R) injury and post-ischemic cardiac remodeling. These mechanisms include metabolic alterations, ROS overproduction, inflammation, autophagy deregulation and mitochondrial dysfunction. This review article discusses the most recent evidence regarding the molecular basis of myocardial ischemic injury and the new potential therapeutic interventions for boosting cardioprotection and attenuating cardiac remodeling.

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Publisher

MDPI AG,MDPI

Subject

Adenosine triphosphate

/ Autophagy

/ Cardiomyocytes

/ Cardiovascular disease

/ Cardiovascular diseases

/ Congestive heart failure

/ Coronary vessels