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We study a neutral honeycomb array of acceptors in a Group IV semiconductor. Tight-binding exhibits a band gap from different hopping of angular momentum ± 3 2 and ± 1 2 , forming a topological insulator (TI). The hopping is even under separate reversal of spatial and spin motions, unlike spin–orbit coupling. The TI is robust to Coulomb interactions and realistic electronic structure calculations show it survives for a range of spacings and distortions commensurate with the silicon growth surface, but has an instability towards spin density wave formation at large separations.

Prompt emissions from TeV blazars pair produce off the extragalactic background light and the highly energetic resulting pair beams then cascade through inverse Compton scattering to give rise to secondary gamma-rays. Such reprocessed cascade emission that can be associated with individual blazar sources has not been detected thus far. The absence of pair halos around these sources, along with the non-observation of isotropic gamma-ray background excess, seems to suggest that collective plasma effects, such as beam-plasma instabilities, can play a crucial role in alleviating this GeV-TeV tension by transferring the energy from the pair beams into the background plasma of the intergalactic medium (IGM). This has profound implications not only for TeV astrophysics, but also the strength of the intergalactic magnetic field and properties of dark matter (DM). A direct consequence of the instability losses and IGM heating is the modification of thermal history at late times, which suppresses structure formation particularly in baryonically underdense regions, potentially holding a clue towards resolving the small-scale crisis in cosmology. In a blazar-heated universe, the observation of dwarf galaxies and Lyman-\\(\\alpha\\) measurements present a favoured mass range for DM candidates such as light axion-like particles.

98% of T cells reside in tissues, yet nearly all human T cell analyses are performed from peripheral blood. We single-cell sequenced 5.7 million T cells from ten donors' autologous blood and tonsils and sought to answer key questions about T cell receptor biology previously unanswerable by smaller-scale experiments. We identified distinct clonal expansions and distributions in blood compared to tonsils, with surprisingly low (1-7%) clonal sharing. These few shared clones exhibited divergent phenotypes across bodily sites. Analysis of antigen-specific CD8 T cells revealed location as a main determinant of frequency, phenotype, and immunodominance. Finally, diversity estimates from the tissue recalibrates current repertoire diversity estimates, and we provide a refined estimate of whole-body repertoire. Given the tissue-restricted nature of T cell phenotypes, functions, differentiation, and clonality revealed by this dataset, we conclude that tissue analyses are crucial for accurate repertoire analysis and monitoring changes after perturbing therapies.