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We describe monkeypox virus (MPXV) transmission from a patient to a healthcare worker through needlestick injury. A lesion appeared at the inoculation site 5 days after injury. Blood tested MPXV-positive by PCR before symptoms worsened; blood remained MPXV-positive at discharge 19 days after symptom onset. Postexposure prophylaxis could prevent potential MPXV bloodborne transmission.

Another study assessed whether HFNO or continuous positive airway pressure was associated with increased levels of viral RNA in air samples compared to standard oxygen therapy.9 Only 30% participants had at least 1 positive or suspected positive result and they showed that Ct values were much higher than a paired nasopharyngeal sample. [...]we did not sample surfaces as another surrogate of environmental spread, although fomites are likely not the main transmission route. [...]air samples were collected mainly from patients in the second week of disease and may have been less infective; nevertheless, this timing is representative of patients with the timing of respiratory failure onset and potential need for NIRS.

Visceral leishmaniasis (VL) is mainly caused by Leishmania (Leishmania) donovani and Leishmania (L.) infantum; however, other Leishmania species have been associated with VL. We report a case of a patient simultaneously diagnosed with VL caused by Leishmania (L.) amazonensis and Hodgkin’s lymphoma. After treatment with liposomal amphotericin B and chemotherapy, the patient presented a clinical cure. This case report reinforces the hypothesis that other Leishmania species can cause visceral lesions mainly related to immunosuppression.