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18 result(s) for "Corey, Shana"
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Barack Obama : out of many, one
In simple language, newly independent readers can learn how the lessons and love of Obama's mother and grandparents shaped him, and how he turned his sadness from his childhood into a positive, driving force of finding a place in the world and in the history books.
The Secret Subway
Bush reviews The Secret Subway by Shana Corey and illustrated by Red Nose Studio.
You forgot your skirt Ameila Bloomer
Amelia Bloomer was not a proper lady. In fact, Amelia Bloomer thought proper ladies were silly. She thought it was silly that proper ladies were not allowed to vote. So she tried to change the laws so that they could. Then one day, Amelia's friend Elizabeth Cady Stanton came to visit. Elizabeth brought her cousin Libby with her. Libby looked remarkable! She was even more improper than Amelia because Libby was not wearing a dress! The silliest thing of all, thought Amelia Bloomer, was the way proper ladies were supposed to dress. Their dresses were so heavy, wearing them was like carting around ... a dozen bricks! What was proper about that?
Boats!
Illustrations and simple rhyming text present all kinds of boats--yachts, toy boats, tugs, ferry boats, and more.
You Forgot Your Skirt, Amelia Bloomer!
A melia Bloomer was not a proper lady. In fact, Amelia Bloomer thought proper ladies were silly.
Michelle Obama : first lady, going higher
\"Michelle Obama is a lot like YOU! She grew up on the South Side of Chicago with her brother, Craig. She rode her bike and played freeze tag with her friends. And she and her family ate dinner together every night! Michelle's parents taught her to work hard and not let anyone or anything stand in her way. That work ethic has propelled her through her whole life--through her magnet high school, her college years at Princeton, and Harvard Law School. Her parents also taught her to reach back and help others once she found success, evidence of which is everywhere in her work as First Lady of the United States and beyond\"--Provided by publisher.
Trends in cognitive outcomes in middle-aged Americans across three birth cohorts
Middle age is a key life course period for targeting modifiable risk factors for late-life cognitive decline. Although the prevalence of chronic conditions that are risk factors for cognitive impairment has increased in middle-aged adults since the 1990s, little is known about corresponding trends in cognitive trajectories and incidence of cognitive impairment. We conducted a cohort study of 7,852 participants ages 50-56 enrolled from 1998-2010 in the Health and Retirement Study (HRS) without cognitive impairment at baseline. Participants were followed biennially to examine cognitive trajectories and new-onset cognitive impairment by age 65, based on the HRS cognitive test battery. We used mixed effects models to examine change in cognitive scores and Cox regression models to compare incidence of cognitive impairment no dementia (CIND) across three birth cohorts (1942-1947, \"War Babies\"; 1948-1953, \"Early Baby Boomers\"; 1954-1959, \"Mid Baby Boomers\"). Mid Boomers had lower baseline cognitive scores compared to earlier cohorts but a slower rate of cognitive decline. Hazards of CIND were higher among White Early and Mid Boomers compared to War Babies in the first half of follow-up, but lower in the second half. In both trajectory and incidence analyses, those with lower educational attainment and racial/ethnic minorities were at higher risk for worse cognitive outcomes. Findings show mixed trends in cognitive outcomes among middle-aged Americans. Overall, Mid Boomers had lower baseline cognitive scores but slower decline compared to earlier cohorts. However, disparities in cognitive outcomes persisted among those with lower educational attainment and racial/ethnic minorities.
Contributions of neighborhood social environment and air pollution exposure to Black-White disparities in epigenetic aging
Racial disparities in many aging-related health outcomes are persistent and pervasive among older Americans, reflecting accelerated biological aging for Black Americans compared to White, known as weathering. Environmental determinants that contribute to weathering are poorly understood. Having a higher biological age, measured by DNA methylation (DNAm), than chronological age is robustly associated with worse age-related outcomes and higher social adversity. We hypothesize that individual socioeconomic status (SES), neighborhood social environment, and air pollution exposures contribute to racial disparities in DNAm aging according to GrimAge and Dunedin Pace of Aging methylation (DPoAm). We perform retrospective cross-sectional analyses among 2,960 non-Hispanic participants (82% White, 18% Black) in the Health and Retirement Study whose 2016 DNAm age is linked to survey responses and geographic data. DNAm aging is defined as the residual after regressing DNAm age on chronological age. We observe Black individuals have significantly accelerated DNAm aging on average compared to White individuals according to GrimAge (239%) and DPoAm (238%). We implement multivariable linear regression models and threefold decomposition to identify exposures that contribute to this disparity. Exposure measures include individual-level SES, census-tract-level socioeconomic deprivation and air pollution (fine particulate matter, nitrogen dioxide, and ozone), and perceived neighborhood social and physical disorder. Race and gender are included as covariates. Regression and decomposition results show that individual-level SES is strongly associated with and accounts for a large portion of the disparity in both GrimAge and DPoAm aging. Higher neighborhood deprivation for Black participants significantly contributes to the disparity in GrimAge aging. Black participants are more vulnerable to fine particulate matter exposure for DPoAm, perhaps due to individual- and neighborhood-level SES, which may contribute to the disparity in DPoAm aging. DNAm aging may play a role in the environment “getting under the skin”, contributing to age-related health disparities between older Black and White Americans.
Dementia Care Research and Psychosocial Factors
A parent's well-being is instrumental for the child's safety, health and enrichment and these factors are all known to contribute to poorer late-life health outcomes. Epigenetic age, measured using DNA methylation, represents a biological feature of aging that reflects the extent to which an individual is accelerated or decelerated relative to their chronological age. Here we evaluate how parental relationship type and well-being associate with later life epigenetic age. Cognitively unimpaired older (>65 years) adults (N = 152) completed the Penn Alzheimer's Disease Research Center's Life Experience Survey. Questions inquired about relationship type (i.e., mother, father, aunt etc.) and overall well-being of up to three childhood caregivers. Ratings were from 0-100 with higher being better. Participants also completed measures of socioeconomic status and sociodemographic data. DNA methylation was measured using genomic blood and the Illumina EPIC array to compute Horvath and Levine epigenetic clocks. Linear regression models examined effects of parental relationship type and parental well-being ratings on age acceleration in bivariate models, which permitted examination of single parents and types of dyads (i.e., mother-father, mother-grandmother) and trivariate models, which allowed each parent relationship type to covary independently. Multivariable models controlled for potential confounding from socioeconomic status, adverse childhood events, and key demographic factors. Father relationship type showed a statistical trend toward decelerated aging (Horvath, β=3.5, 95%CI -0.02 to 7.0). In bivariate models, better parental well-being was associated with decelerated aging (Levin, β=0.14, 95%CI 0.02, 0.24). Secondary parent well-being showed no effect. In trivariate models, better primary parent well-being was associated with decelerated aging (Levin, β=0.14, 95%CI 0.02, 0.24; Horvath, β=0.09, 95%CI 0.03, 0.10). Secondary parent well-being higher than the primary parent's was associated with accelerated aging (Levin, β=-0.08, 95%CI -0.2 to 0.02, p = 0.05; Horvath, β=-0.09, 95%CI -0.1, -0.03). These associations remained in multivariable models. Our findings provide evidence for protective and negative biological aging outcomes associated with primary compared to secondary parental well-being. Thus, it is important to consider social context on aging outcomes. The findings underscore the import of caregivers in the health of older adults. This research line may inform population-level health interventions.