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The OceanField project is an integrated field-work and classroom-based course offered to first year Master students in Marine Geosciences (at the European Institute for Marine Studies IUEM - University of Brest), creating a synergy between (1) geology field class, (2) photogrammetric data acquisition and (3) data processing to produce digital terrain models, enabling the immersive experience to be extended in a digital working environment once back in class. In this way, the students experiment different approaches for observing and analysing the structure, geometry and nature of a past oceanic domain in the Alps, and gain an understanding of how it works (from its birth to its disappearance). At the same time, participating in the acquisition and processing of photogrammetric data, students acquire new technical skills. By not only being immersed in the virtual environment, but also contributing to its creation, students are involved in the various stages of the data lifecycle. As a result, they become more aware of multiscale data quality and of the opportunities offered by virtual environment accuracy.

Oceanic crust formed at slow-spreading ridges is currently subducted in only a few places on Earth and the tectonic and seismogenic imprint of the slow-spreading process is poorly understood. Here we present seismic and bathymetric data from the Northeastern Lesser Antilles Subduction Zone where thick sediments enable seismic imaging to greater depths than in the ocean basins. This dataset highlights a pervasive tectonic fabric characterized by closely spaced sequences of convex-up Ridgeward-Dipping Reflectors, which extend down to about 15 km depth with a 15-to-40° angle. We interpret these reflectors as discrete shear planes formed during the early stages of exhumation of magma-poor mantle rocks at an inside corner of a Mid-Atlantic Ridge fracture zone. Closer to the trench, plate bending could have reactivated this tectonic fabric and enabled deep fluid circulation and serpentinization of the basement rocks. This weak serpentinized basement likely explains the very low interplate seismic activity associated with the Barbuda-Anegada margin segment above.

The 26 December 2004 Sumatra earthquake (Mw = 9.1) initiated around 30 km depth and ruptured 1300 km of the Indo‐Australian–Sunda plate boundary. During the Sumatra‐OBS (ocean bottom seismometer) survey, a wide‐angle seismic profile was acquired across the epicentral region. A seismic velocity model was obtained from combined travel time tomography and forward modeling. Together with reflection seismic data from the SeaCause II cruise, the deep structure of the source region of the great earthquake is revealed. Four to five kilometers of sediments overlie the oceanic crust at the trench, and the subducting slab can be imaged down to a depth of 35 km. We find a crystalline backstop 120 km from the trench axis, below the fore‐arc basin. A high‐velocity zone at the lower landward limit of the ray‐covered domain, at 22 km depth, marks a shallow continental Moho, 170 km from the trench. The deep structure obtained from the seismic data was used to construct a thermal model of the fore arc in order to predict the limits of the seismogenic zone along the plate boundary fault. Assuming 100°–150°C as its updip limit, the seismogenic zone is predicted to begin 5–30 km from the trench. The downdip limit of the 2004 rupture as inferred from aftershocks is within the 350°–450°C temperature range, but this limit is 210–250 km from the trench axis and is much deeper than the fore‐arc Moho. The deeper part of the rupture occurred along the contact between the mantle wedge and the downgoing plate.

The nature of the Ionian Sea crust has been the subject of scientific debate for more than 30 years, mainly because seismic imaging of the deep crust and upper mantle of the Ionian Abyssal Plain (IAP) has not been conclusive to date. The IAP is sandwiched between the Calabrian and Hellenic subduction zones in the central Mediterranean. A NNE–SSW-oriented 131 km long seismic refraction and wide-angle reflection profile, consisting of eight ocean bottom seismometers and hydrophones, was acquired in 2014. The profile was designed to univocally confirm the proposed oceanic nature of the IAP crust as a remnant of the Tethys and to confute its interpretation as a strongly thinned part of the African continental crust. A P-wave velocity model developed from travel-time forward modelling is refined by gravimetric data and synthetic modelling of the seismic data. A roughly 6–7 km thick crust with velocities ranging from 5.1 to 7.2 km s−1, top to bottom, can be traced throughout the IAP. In the vicinity of the Medina seamounts at the southern IAP boundary, the crust thickens to about 9 km and seismic velocities decrease to 6.8 km s−1 at the crust–mantle boundary. The seismic velocity distribution and depth of the crust–mantle boundary in the IAP document its oceanic nature and support the interpretation of the IAP as a remnant of the Tethys lithosphere with the Malta Escarpment as a transform margin and a Tethys opening in the NNW–SSE direction.

It is widely accepted that the Central and Eastern Mediterranean are remnants of the Neo‐Tethys. However, the orientation and timing of spreading of this domain remain controversial. Here, we present time migrated and pre‐stack depth migrated NW‐SE oriented Archimede (1997) lines together with the PrisMed01 (1993) profile to constrain the evolution of the Ionian basin. Our interpretation allows us to identify a large‐scale set of SW‐NE striking reverse faults beneath the Ionian Abyssal Plain. These primarily NW vergent faults have a characteristic spacing of 10 to 20 km and a dip ranging from 60 to 65°. Following very recent paleogeographic reconstructions, we propose that the set of N°55 features initially formed as normal faults during NW‐SE trending seafloor spreading of the Ionian basin after its late Triassic‐early Jurassic rifting. Based on geometric comparisons with the intraplate deformation observed beneath the Central Indian Ocean, we show that the inherited oceanic normal faults were reactivated under compression as reverse faults. Well‐developed Tortonian syntectonic basins developed NW of the major faults and the base of the Messinian evaporites (Mobile Unit) is slightly folded by the activity of the faults. We show that 3–4 km of total shortening occurs over a 80 km wide area beneath the Ionian Abyssal Plain, resulting in a bulk shortening of 3.5–5%. We propose a link between the Tortonian‐early Messinian inversion of the fault pattern and a plate tectonic reorganization prior to the main phase of back‐arc opening of the Tyrrhenian domain. Key Points Seismic profiles image the structure and deformation beneath the Ionian sea NW vergent thrust faults affect pre‐ and post‐Messinian strata Faults represent reactivated sea‐floor fabric from Tethyan rifting and spreading

After the January 12, 2010, Haiti earthquake, we deployed a mainly offshore temporary network of seismologic stations around the damaged area. The distribution of the recorded aftershocks, together with morphotectonic observations and mainshock analysis, allow us to constrain a complex fault pattern in the area. Almost all of the aftershocks have a N‐S compressive mechanism, and not the expected left‐lateral strike‐slip mechanism. A first‐order slip model of the mainshock shows a N264°E north‐dipping plane, with a major left‐lateral component and a strong reverse component. As the aftershock distribution is sub‐parallel and close to the Enriquillo fault, we assume that although the cause of the catastrophe was not a rupture along the Enriquillo fault, this fault had an important role as a mechanical boundary. The azimuth of the focal planes of the aftershocks are parallel to the north‐dipping faults of the Transhaitian Belt, which suggests a triggering of failure on these discontinuities. In the western part, the aftershock distribution reflects the triggering of slip on similar faults, and/or, alternatively, of the south‐dipping faults, such the Trois‐Baies submarine fault. These observations are in agreement with a model of an oblique collision of an indenter of the oceanic crust of the Southern Peninsula and the sedimentary wedge of the Transhaitian Belt: the rupture occurred on a wrench fault at the rheologic boundary on top of the under‐thrusting rigid oceanic block, whereas the aftershocks were the result of the relaxation on the hanging wall along pre‐existing discontinuities in the frontal part of the Transhaitian Belt. Key Points Active fault identification for the Jan.12, 2010 Haiti earthquake Ocean Bottom Seismometer ued for aftershocks studies Transpressive mechanism for the Jan.12, 2010 Haiti earthquake

The 26 December 2004 Sumatra earthquake (Mw = 9.1) initiated around 30 km depth and ruptured 1300 km of the Indo-Australian Sunda plate boundary. During the Sumatra OBS (ocean bottom seismometer) survey, a wide angle seismic profile was acquired across the epicentral region. A seismic velocity model was obtained from combined travel time tomography and forward modeling. Together with reflection seismic data from the SeaCause II cruise, the deep structure of the source region of the great earthquake is revealed. Four to five kilometers of sediments overlie the oceanic crust at the trench, and the subducting slab can be imaged down to a depth of 35 km. We find a crystalline backstop 120 km from the trench axis, below the fore arc basin. A high velocity zone at the lower landward limit of the raycovered domain, at 22 km depth, marks a shallow continental Moho, 170 km from the trench. The deep structure obtained from the seismic data was used to construct a thermal model of the fore arc in order to predict the limits of the seismogenic zone along the plate boundary fault. Assuming 100C-150C as its updip limit, the seismogenic zone is predicted to begin 530 km from the trench. The downdip limit of the 2004 rupture as inferred from aftershocks is within the 350C 450C temperature range, but this limit is 210-250 km from the trench axis and is much deeper than the fore arc Moho. The deeper part of the rupture occurred along the contact between the mantle wedge and the downgoing plate.

Metabolic Dysfunction-Associated Steatotic Liver Disease (MASLD) is a chronic liver disease affecting 25% of the European population, with rising global incidence. Liver damage includes ballooning, steatosis, inflammation and fibrosis. Associated brain disorders include sleep, cognitive issues, anxiety, and depression. While neurological complications in advanced MASLD are well documented, early cerebral manifestations remain largely unexplored. This study aimed at developing an MASLD rat model to assess the onset of early brain damage, focusing on impairments of the circadian cycle rhythm and associated neuroinflammation. Sprague Dawley rats were divided into two groups: one received a high-fat, high-cholesterol (HFHC) diet for 90 days, while the other received a standard diet. Histological analysis showed significant hepatic steatosis, ballooning, and inflammation in the HFHC group ( p  < 0.01). These lesions correlated with elevated hepatic triglycerides ( p  < 0.01), increased Alanine Aminotransferase, Aspartate Aminotransferase, total cholesterol, and low-density lipoprotein, alongside decreased plasma high-density lipoprotein. Behavioural analysis using activity wheels revealed that the HFHC rats steadily maintained their activity level during the rest periods when compared with controls ( p  < 0.05). This behavioural alteration occurred alongside neuroinflammation, demonstrated by changes in the expression of 36 and 17 inflammatory mediators in the cerebellum and frontal cortex respectively. These changes were associated with an increase in the expression of glial cell markers ( Aif1 and Gfap genes) and an increase in the number of microglial cells, affecting the frontal cortex and cerebellum differently. This rat model of early MASLD shows circadian rhythm disturbances, which could reflect sleep disorders in humans. These early brain disturbances specific to MASLD, which occur before the symptoms of liver disease become clinically apparent, could therefore be used as an early diagnosis marker for MASLD patients.

Virus-induced plant diseases in cultivated plants cause important damages in yield. Although the mechanisms of virus infection are intensely studied at the cell biology level, only little is known about the molecular dialog between the invading virus and the host genome. Here we describe a combinatorial genome-wide approach to identify networks of sRNAs-guided post-transcriptional regulation within local Turnip mosaic virus (TuMV) infection sites in Brassica napus leaves. We show that the induction of host-encoded, virus-activated small interfering RNAs (vasiRNAs) observed in virus-infected tissues is accompanied by site-specific cleavage events on both viral and host RNAs that recalls the activity of small RNA-induced silencing complexes (RISC). Cleavage events also involve virus-derived siRNA (vsiRNA)–directed cleavage of target host transcripts as well as cleavage of viral RNA by both host vasiRNAs and vsiRNAs. Furthermore, certain coding genes act as virus-activated regulatory hubs to produce vasiRNAs for the targeting of other host genes. The observations draw an advanced model of plant-virus interactions and provide insights into the complex regulatory networking at the plant-virus interface within cells undergoing early stages of infection. Pitzalis et al. use replicative RNAseq, small RNA (sRNA)seq, and parallel analysis of RNA ends (PARE)seq analysis to identify networks of sRNAs-guided post-transcriptional regulation within local Turnip mosaic virus infection sites. This study provides insights into the complex regulatory networking at the plantvirus interface within cells undergoing early stages of infection.