Search Results Heading

MBRLSearchResults

mbrl.module.common.modules.added.book.to.shelf
Title added to your shelf!
View what I already have on My Shelf.
Oops! Something went wrong.
Oops! Something went wrong.
While trying to add the title to your shelf something went wrong :( Kindly try again later!
Are you sure you want to remove the book from the shelf?
Oops! Something went wrong.
Oops! Something went wrong.
While trying to remove the title from your shelf something went wrong :( Kindly try again later!
    Done
    Filters
    Reset
  • Discipline
      Discipline
      Clear All
      Discipline
  • Is Peer Reviewed
      Is Peer Reviewed
      Clear All
      Is Peer Reviewed
  • Reading Level
      Reading Level
      Clear All
      Reading Level
  • Content Type
      Content Type
      Clear All
      Content Type
  • Year
      Year
      Clear All
      From:
      -
      To:
  • More Filters
      More Filters
      Clear All
      More Filters
      Item Type
    • Is Full-Text Available
    • Subject
    • Country Of Publication
    • Publisher
    • Source
    • Target Audience
    • Language
    • Place of Publication
    • Contributors
    • Location
628 result(s) for "Paxton, Robert"
Sort by:
تشريح الفاشية
يتناول هذا الكتاب : \"الفاشية\"ما هي الفاشية ؟ اقترح العديد من المؤلفين تعريفات، لكن معظمهم فشلوا في تجاوز الملخص. يجيب المؤرخ المحترم روبرت أو باكستون على هذا السؤال لأول مرة من خلال التركيز على الخرسانة : ما فعله الفاشيون، بدلا من ما قالوا. من أول العصابات النظامية العنيفة التي تضرب \"أعداء الدولة\"، من خلال صعود موسوليني إلى السلطة، إلى التطرف الفاشي الألماني في الحرب العالمية الثانية، يظهر باكستون بوضوح لماذا جاء الفاشيون إلى السلطة في بعض البلدان وليس في غيرها، ويستكشف ما إذا كان يمكن للفاشية موجودة خارج الإعداد الأوروبي في أوائل القرن العشرين الذي ظهرت فيه. سيكون لتشريح الفاشية تأثير دائم على فهمنا للتاريخ الأوروبي الحديث، تماما مثلما أعادت فيشي فرنسا الكلاسيكية في باكستون تحديد رؤيتنا للحرب العالمية الثانية. استنادا إلى عمر البحث، يحول هذا الكتاب الهام والمهم معرفتنا بالفاشية-\"الابتكار السياسي الرئيسي في القرن العشرين ومصدر الكثير من آلامها\".
Symbionts as Major Modulators of Insect Health: Lactic Acid Bacteria and Honeybees
Lactic acid bacteria (LAB) are well recognized beneficial host-associated members of the microbiota of humans and animals. Yet LAB-associations of invertebrates have been poorly characterized and their functions remain obscure. Here we show that honeybees possess an abundant, diverse and ancient LAB microbiota in their honey crop with beneficial effects for bee health, defending them against microbial threats. Our studies of LAB in all extant honeybee species plus related apid bees reveal one of the largest collections of novel species from the genera Lactobacillus and Bifidobacterium ever discovered within a single insect and suggest a long (>80 mya) history of association. Bee associated microbiotas highlight Lactobacillus kunkeei as the dominant LAB member. Those showing potent antimicrobial properties are acquired by callow honey bee workers from nestmates and maintained within the crop in biofilms, though beekeeping management practices can negatively impact this microbiota. Prophylactic practices that enhance LAB, or supplementary feeding of LAB, may serve in integrated approaches to sustainable pollinator service provision. We anticipate this microbiota will become central to studies on honeybee health, including colony collapse disorder, and act as an exemplar case of insect-microbe symbiosis.
Urban areas as hotspots for bees and pollination but not a panacea for all insects
Urbanisation is an important global driver of biodiversity change, negatively impacting some species groups whilst providing opportunities for others. Yet its impact on ecosystem services is poorly investigated. Here, using a replicated experimental design, we test how Central European cities impact flying insects and the ecosystem service of pollination. City sites have lower insect species richness, particularly of Diptera and Lepidoptera, than neighbouring rural sites. In contrast, Hymenoptera, especially bees, show higher species richness and flower visitation rates in cities, where our experimentally derived measure of pollination is correspondingly higher. As well as revealing facets of biodiversity (e.g. phylogenetic diversity) that correlate well with pollination, we also find that ecotones in insect-friendly green cover surrounding both urban and rural sites boost pollination. Appropriately managed cities could enhance the conservation of Hymenoptera and thereby act as hotspots for pollination services that bees provide to wild flowers and crops grown in urban settings. Pollinators can persist in urban areas despite little natural habitat. Here the authors compare insect pollinators and pollination inside and outside of German cities, showing that urban areas have high diversity of bees but not other insects, and high pollination provisioning, relative to rural sites.
The novel insecticides flupyradifurone and sulfoxaflor do not act synergistically with viral pathogens in reducing honey bee (Apis mellifera) survival but sulfoxaflor modulates host immunocompetence
Through laboratory experiments in which treatments were administered singly or in combination to individual insects, we recorded harmful effects of FPF and pathogens on honey bee survival and immune gene expression. Though we found no evidence of synergistic interactions among stressors on either honey bee survival or viral load, the combined treatment SULF and DWV‐B led to a synergistic up‐regulation of dicer‐like gene expression. We conclude that common viral pathogens pose a major threat to honey bees whilst co‐exposure to these novel nACHR insecticides does not significantly exacerbate viral impacts on host survival in the laboratory. Summary The decline of insect pollinators threatens global food security. A major potential cause of decline is considered to be the interaction between environmental stressors, particularly between exposure to pesticides and pathogens. To explore pesticide–pathogen interactions in an important pollinator insect, the honey bee, we used two new nicotinic acetylcholine receptor agonist insecticides (nACHRs), flupyradifurone (FPF) and sulfoxaflor (SULF), at sublethal and field‐realistic doses in a fully crossed experimental design with three common viral honey bee pathogens, Black queen cell virus (BQCV) and Deformed wing virus (DWV) genotypes A and B. Through laboratory experiments in which treatments were administered singly or in combination to individual insects, we recorded harmful effects of FPF and pathogens on honey bee survival and immune gene expression. Though we found no evidence of synergistic interactions among stressors on either honey bee survival or viral load, the combined treatment SULF and DWV‐B led to a synergistic upregulation of dicer‐like gene expression. We conclude that common viral pathogens pose a major threat to honey bees, while co‐exposure to these novel nACHR insecticides does not significantly exacerbate viral impacts on host survival in the laboratory.
Experimental cross species transmission of a major viral pathogen in bees is predominantly from honeybees to bumblebees
Cross-species transmission of a pathogen from a reservoir to a recipient host species, spillover, can have major impacts on biodiversity, domestic species and human health. Deformed wing virus (DWV) is a panzootic RNA virus in honeybees that is causal in their elevated colony losses, and several correlative field studies have suggested spillover of DWV from managed honeybees to wild bee species such as bumblebees. Yet unequivocal demonstration of DWV spillover is lacking, while spillback, the transmission of DWV from a recipient back to the reservoir host, is rarely considered. Here, we show in fully crossed laboratory experiments that the transmission of DWV (genotype A) from honeybees to bumblebees occurs readily, yet we neither detected viral transmission from bumblebees to honeybees nor onward transmission from experimentally infected to uninoculated bumblebees. Our results support the potential for viral spillover from honeybees to other bee species in the field when robbing resources from heterospecific nests or when visiting the same flowers. They also underscore the importance of studies on the virulence of DWV in wild bee species so as to evaluate viral impact on individual and population fitness as well as viral adaption to new host species.
Urbanization is associated with shifts in bumblebee body size, with cascading effects on pollination
Urbanization is a global phenomenon with major effects on species, the structure of community functional traits and ecological interactions. Body size is a key species trait linked to metabolism, life‐history and dispersal as well as a major determinant of ecological networks. Here, using a well‐replicated urban–rural sampling design in Central Europe, we investigate the direction of change of body size in response to urbanization in three common bumblebee species, Bombus lapidarius, Bombus pascuorum and Bombus terrestris, and potential knock‐on effects on pollination service provision. We found foragers of B. terrestris to be larger in cities and the body size of all species to be positively correlated with road density (albeit at different, species‐specific scales); these are expected consequences of habitat fragmentation resulting from urbanization. High ambient temperature at sampling was associated with both a small body size and an increase in variation of body size in all three species. At the community level, the community‐weighted mean body size and its variation increased with urbanization. Urbanization had an indirect positive effect on pollination services through its effects not only on flower visitation rate but also on community‐weighted mean body size and its variation. We discuss the eco‐evolutionary implications of the effect of urbanization on body size, and the relevance of these findings for the key ecosystem service of pollination.
A microbiome silver bullet for honey bees
A genetically engineered honey bee gut bacterium knocks down two major bee threats The western honey bee ( Apis mellifera ) brings tangible benefits to humans as an important pollinator and insights into social evolution as a model organism. Yet, despite close scientific scrutiny, it is under global threat from a range of stressors ( 1 ) that are unlikely to diminish with global change. Chief among these are pests and pathogens, remedies to which are either ineffective, short-term, expensive, or impractical. On page 573 of this issue, Leonard et al. ( 2 ) reveal a hidden microbiological key to fight these pests and pathogens: genetically modified honey bee gut bacteria tailored to induce host RNA interference (RNAi)–based defense ( 3 ) that is effective, long-term, potentially cheap, and easy to apply. This important approach may not only provide a solution to many of the honey bee's woes, it also offers a new functional genomic toolkit with which to dissect the molecular intricacies of honey bees and their societies.
The virulent, emerging genotype B of Deformed wing virus is closely linked to overwinter honeybee worker loss
Bees are considered to be threatened globally, with severe overwinter losses of the most important commercial pollinator, the Western honeybee, a major concern in the Northern Hemisphere. Emerging infectious diseases have risen to prominence due to their temporal correlation with colony losses. Among these is Deformed wing virus (DWV), which has been frequently linked to colony mortality. We now provide evidence of a strong statistical association between overwintering colony decline in the field and the presence of DWV genotype-B (DWV-B), a genetic variant of DWV that has recently been shown to be more virulent than the original DWV genotype-A. We link the prevalence of DWV-B directly to a quantitative measure of overwinter decline (workforce mortality) of honeybee colonies in the field. We demonstrate that increased prevalence of virus infection in individual bees is associated with higher overwinter mortality. We also observed a substantial reduction of infected colonies in the spring, suggesting that virus-infected individuals had died during the winter. Our findings demonstrate that DWV-B, plus possible A/B recombinants exhibiting DWV-B at PCR primer binding sites, may be a major cause of elevated overwinter honeybee loss. Its potential emergence in naïve populations of bees may have far-reaching ecological and economic impacts.
Urban fragmentation leads to lower floral diversity, with knock-on impacts on bee biodiversity
Bees and flowering plants are two closely interacting groups of organisms. Habitat loss and fragmentation associated with urbanisation are major threats to both partners. Yet how and why bee and floral richness and diversity co-vary within the urban landscape remain unclear. Here, we sampled bees and flowering plants in urban green spaces to investigate how bee and flowering plant species richness, their phylogenetic diversity and pollination-relevant functional trait diversity influence each other in response to urban fragmentation. As expected, bee abundance and richness were positively related to flowering plant richness, with bee body size (but not bee richness and diversity) increasing with nectar-holder depth of flowering plants. Causal modelling indicated that bottom-up effects dictated patterns of bee-flower relationships, with urban fragmentation diminishing flowering plants richness and thereby indirectly reducing bee species richness and abundance. The close relationship between bees and flowering plants highlights the risks of their parallel declines in response to land-use change within the urban landscape.
Elevated virulence of an emerging viral genotype as a driver of honeybee loss
Emerging infectious diseases (EIDs) have contributed significantly to the current biodiversity crisis, leading to widespread epidemics and population loss. Owing to genetic variation in pathogen virulence, a complete understanding of species decline requires the accurate identification and characterization of EIDs. We explore this issue in the Western honeybee, where increasing mortality of populations in the Northern Hemisphere has caused major concern. Specifically, we investigate the importance of genetic identity of the main suspect in mortality, deformed wing virus (DWV), in driving honeybee loss. Using laboratory experiments and a systematic field survey, we demonstrate that an emerging DWV genotype (DWV-B) is more virulent than the established DWV genotype (DWV-A) and is widespread in the landscape. Furthermore, we show in a simple model that colonies infected with DWV-B collapse sooner than colonies infected with DWV-A. We also identify potential for rapid DWV evolution by revealing extensive genome-wide recombination in vivo. The emergence of DWV-B in naive honeybee populations, including via recombination with DWV-A, could be of significant ecological and economic importance. Our findings emphasize that knowledge of pathogen genetic identity and diversity is critical to understanding drivers of species decline.