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This paper investigates the relationship between environmental and social disclosure with earnings persistence as a proxy of earnings quality. This research is based on the secondary data obtained from the final reports of the listed firms of Malaysia. The empirical analysis applied on 136 listed firms of Bursa Malaysia throughout 2011 to 2018 with the observation of 1088. The measure of earnings persistence is derived from the panel data based on the regression of the time series model. The results of our study indicate that environmental and social disclosures are positively correlated with earnings persistence. This means that the more the level of commitment a firm shows with environmental and social responsibility, the more persistent earnings it carries. Moreover, earnings persistence is the proxy of earnings quality, and it is desirable by every investor.

In this research, we planned and conducted estimations for developing a pilot-scale Clean Development Mechanism (CDM) project for group plant activities in the Vietnam electricity/energy sector. The overall aim of this paper is to assess the power generation potential of rice husk power plants in the Mekong Delta. We intend to set up a rice husk energy balance flowchart for the whole Mekong River Delta in the year 2021 and suggest policies that can be used for the power generation of unused rice husk, to avoid having them pollute rivers and canals. We put forward a safe and environmentally friendly solution to thoroughly minimize the current serious pollution of rivers and canals in the Mekong River Delta caused by the increasing quantity of unused rice husk. The results of this paper are based on the estimation of electricity potential of a group of rice husk power development plants in the Mekong River Delta with a capacity of 11 MW per plant, including carbon dioxide emission reductions (CERs) and CER credits, along with estimations of their economic criteria (NPV, B/C, IRR), both W/CDM and W/O CDM.

The current research investigates the role of public-private partnership investment (PPPI) and ecological innovation (ECO) along with economic growth on the environmental abatement (i.e., carbon dioxide emission, particulate matters 2.5) in the USA. Quantile autoregressive lagged (QARDL) method was employed during the study period of 1990-2015. The study findings confirm that under long-run estimation, GDP and PPPI are causing more environmental abatement in the form of CO2 emission and haze pollution like PM2.5. The factors like ecological innovation and GDP2 are playing their role towards lowering the CO2 emission and PM2.5 under different quantiles. Furthermore, it is observed that under short-run estimation, past values of the carbon emissions and PM2.5 have their significant and positive relationship with their current values. Besides, the findings through Wald test estimation confirm that parameter constancy of the speed of adjustment parameter is rejected at 1% significance level for the CO2 emission and haze pollution like PM2.5 in USA. Besides, present study also provides some policy implications.

Distinguishing the significance of business environments for technical small and medium-sized enterprises (SMEs), this study examines the connection between business environments, GDP growth, and SMEs' financing choices in Vietnam. The study considered the agency theory as a theoretical base to explain how information asymmetry between SMEs and lenders influences SMEs' financing choices and encompasses the effects on business environment and GDP growth of Vietnam. For this binary logistic regression, text is applied. Global Entrepreneurship Monitor and World Bank data were analyzed. The findings of the study are robust and showed that SME financing (e.g., formal and informal) under the financial infrastructure and tax regulation may enhance formal credit choice and reduce informal credit choice. This enhances the depth in the business environment of technical SMEs and found significant effects on GDP growth. For the first time, this research examines the impact of information asymmetry and agency theory on restaurant financing choices. The research has significance for industry practitioners and governments interested in SMEs' financial viability. On the recent topicality, study also presents policy implications for key stakeholders.

This paper evaluates the energy poverty of Vietnam by mediating the role of financial development and environmental considerations. Across the globe, billions of individuals live in fuel poverty, failing to access inexpensive and sustainable energy, which is necessary for long-term development. An elevation in power consumption due to an overall increase in heat and short periods of extreme heat exacerbates global warming. The goal of this research is to look at how climate change is affecting energy poverty in Vietnam. This finding (1) demonstrates that temperature shocks have a positive and quantitative impact. (2) The same may be said for “poor income/high cost” figures, which include information on power rates. Similarly, if households use the same amount of power but spend less on other items, the influence will not raise their electricity use. (3) Thermal shocks have been shown to reduce agricultural output in studies. During seasons of low rainfall, for example, higher evaporation and plant water demand can worsen drought and raise total irrigation expenses. Labor productivity is also affected by rising temperatures, particularly in weather-sensitive industries like agriculture. As a result, heat shocks will lower agricultural revenues, worsening energy poverty. Surprisingly, overall income appears to be a little mitigating influence. This might be owing to widespread underreporting of stated income, particularly among persons who rely on agriculture for their livelihood.

Cardioprotection against ischemia/reperfusion injury is still an unmet clinical need. The transient activation of Toll-like receptors (TLRs) has been implicated in cardioprotection, which may be achieved by treatment with blood-derived extracellular vesicles (EVs). However, since the isolation of EVs from blood takes considerable effort, the aim of our study was to establish a cellular model from which cardioprotective EVs can be isolated in a well-reproducible manner. EV release was induced in HEK293 cells with calcium ionophore A23187. EVs were characterized and cytoprotection was assessed in H9c2 and AC16 cell lines. Cardioprotection afforded by EVs and its mechanism were investigated after 16 h simulated ischemia and 2 h reperfusion. The induction of HEK293 cells by calcium ionophore resulted in the release of heterogenous populations of EVs. In H9c2 and AC16 cells, stressEVs induced the downstream signaling of TLR4 and heme oxygenase 1 (HO-1) expression in H9c2 cells. StressEVs decreased necrosis due to simulated ischemia/reperfusion injury in H9c2 and AC16 cells, which was independent of TLR4 induction, but not that of HO-1. Calcium ionophore-induced EVs exert cytoprotection by inducing HO-1 in a TLR4-independent manner.

The title compound, propyl gallate (III), is an important substance popularly used in the food, cosmetic and pharmaceutical industries. Current chemical syntheses of this compound are based on the acylation supported by thionyl chloride, DIC/DMAP or Fischer esterification using a range of homogenous and heterogenous catalysts. In this paper, an efficient, green, straightforward, and economical method for synthesizing propyl gallate using potassium hydrogen sulfate, KHSO4, as the heterogenous acidic catalyst has been developed for the first time. In addition, this paper provides a comprehensive spectral dataset for the title compound, especially the new data on DEPT and 2D NMR (HSQC and HMBC) spectra which are not currently available in the literature.

AP736 was identified as an antimelanogenic drug that can be used for the prevention of melasma, freckles, and dark spots in skin by acting as a suppressor of melanin synthesis and tyrosinase expression. Since macrophage-mediated inflammatory responses are critical for skin health, here we investigated the potential anti-inflammatory activity of AP736. The effects of AP736 on various inflammatory events such as nitric oxide (NO)/prostaglandin (PG) E2 production, inflammatory gene expression, phagocytic uptake, and morphological changes were examined in RAW264.7 cells. AP736 was found to strongly inhibit the production of both NO and PGE2 in lipopolysaccharide- (LPS-) treated RAW264.7 cells. In addition, AP736 strongly inhibited both LPS-induced morphological changes and FITC-dextran-induced phagocytic uptake. Furthermore, AP736 also downregulated the expression of multiple inflammatory genes, such as inducible NO synthase (iNOS), cyclooxygenase- (COX-) 2, and interleukin- (IL-) 1β in LPS-treated RAW264.7 cells. Transcription factor analysis, including upstream signalling events, revealed that both NF-κB and AP-1 were targeted by AP736 via inhibition of the IKK/IκBα and IRAK1/TAK1 pathways. Therefore, our results strongly suggest that AP736 is a potential anti-inflammatory drug due to its suppression of NF-κB-IKK/IκBα and AP-1-IRAK1/TAK1 signalling, which may make AP736 useful for the treatment of macrophage-mediated skin inflammation.

AP736 was identified as an antimelanogenic drug that can be used for the prevention of melasma, freckles, and dark spots in skin by acting as a suppressor of melanin synthesis and tyrosinase expression. Since macrophage-mediated inflammatory responses are critical for skin health, here we investigated the potential anti-inflammatory activity of AP736. The effects of AP736 on various inflammatory events such as nitric oxide (NO)/prostaglandin (PG) E2 production, inflammatory gene expression, phagocytic uptake, and morphological changes were examined in RAW264.7 cells. AP736 was found to strongly inhibit the production of both NO and PGE2 in lipopolysaccharide- (LPS-) treated RAW264.7 cells. In addition, AP736 strongly inhibited both LPS-induced morphological changes and FITC-dextran-induced phagocytic uptake. Furthermore, AP736 also downregulated the expression of multiple inflammatory genes, such as inducible NO synthase (iNOS), cyclooxygenase- (COX-) 2, and interleukin- (IL-) 1β in LPS-treated RAW264.7 cells. Transcription factor analysis, including upstream signalling events, revealed that both NF-κB and AP-1 were targeted by AP736 via inhibition of the IKK/IκBα and IRAK1/TAK1 pathways. Therefore, our results strongly suggest that AP736 is a potential anti-inflammatory drug due to its suppression of NF-κB-IKK/IκBα and AP-1-IRAK1/TAK1 signalling, which may make AP736 useful for the treatment of macrophage-mediated skin inflammation.

The current research investigates the role of public-private partnership investment (PPPI) and ecological innovation (ECO) along with economic growth on the environmental abatement (i.e., carbon dioxide emission, particulate matters 2.5) in the USA. Quantile autoregressive lagged (QARDL) method was employed during the study period of 1990–2015. The study findings confirm that under long-run estimation, GDP and PPPI are causing more environmental abatement in the form of CO2 emission and haze pollution like PM2.5. The factors like ecological innovation and GDP2 are playing their role towards lowering the CO2 emission and PM2.5 under different quantiles. Furthermore, it is observed that under short-run estimation, past values of the carbon emissions and PM2.5 have their significant and positive relationship with their current values. Besides, the findings through Wald test estimation confirm that parameter constancy of the speed of adjustment parameter is rejected at 1% significance level for the CO2 emission and haze pollution like PM2.5 in USA. Besides, present study also provides some policy implications.