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"Starvation"
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Aid airdrops resume in Gaza as starvation deaths rise
Israel resumed airdropping food into Gaza and announced \"tactical pauses\" in fighting on July 27, as deaths from starvation in the enclave worsened.
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World hunger : 10 myths
\"Driven by the question 'Why hunger despite an abundance of food?,' Lappâe and Collins [examine] the myths that prevent us from addressing the root causes of hunger across the globe. [Their book] draws on extensive new research to offer ... insights about tough questions--from climate change and population growth to genetically modified organisms (GMOs) and the role of U.S. foreign aid, and more\"-- Amazon.com
Book
Gazans are being starved to death
After four months of Israeli siege, nearly 1 in 3 people are going multiple days without eating, and deaths are rising from malnutrition.
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Hunger and war : food provisioning in the Soviet Union during World War II
\"Making use of recently released Soviet archival materials, Hunger and War investigates state food supply policy and its impact on Soviet society during World War II. It explores the role of the state in provisioning the urban population, particularly workers, with food, and in feeding the Red army; the medicalization of hunger; hunger in blockaded Leningrad; and civilian mortality from hunger and malnutrition in other home front industrial regions. New research reported here challenges and complicates many of the narratives and counter-narratives about the war. The authors engage such difficult subjects as starvation mortality, bitterness over privation and inequalities in provisioning, and conflicts among state organizations. At the same time, they recognize the considerable role played by the Soviet state in organizing supplies of food to adequately support the military effort and defense production, and in developing policies that promoted social stability amid upheaval. The book makes a significant contribution to scholarship on the Soviet population's experience of World War II as well as to studies of war and famine\"--Provided by publisher.
Book
Fighting famine in North China : state, market, and environmental decline, 1690s-1990s
This monumental work provides a new perspective on the historical significance of famines in China over the past three hundred years. It examines the relationship between the interventionist state policies of the eighteenth-century Qing emperors ('The golden age of famine relief'), the environmental and political crises of the nineteenth and early twentieth centuries (when China was called 'the Land of Famine'), and the ambitions of the Mao era (which tragically led to the greatest famine in human history). In addition to a wide array of documentary sources, the book employs quantitative analysis to measure the economic impact of natural crises, state policies, and markets. In this way, the theories of Qing statesmen that have received much attention in recent scholarship are linked to actual practices and outcomes. Using the Zhili-Hebei region as its focus, the book also reveals the unusual role played by the institutions and policies designed to ensure food security for the capital, Beijing. -- Publisher description.
Book
The long road to leptin
Leptin is an adipose tissue hormone that functions as an afferent signal in a negative feedback loop that maintains homeostatic control of adipose tissue mass. This endocrine system thus serves a critical evolutionary function by protecting individuals from the risks associated with being too thin (starvation) or too obese (predation and temperature dysregulation). Mutations in leptin or its receptor cause massive obesity in mice and humans, and leptin can effectively treat obesity in leptin-deficient patients. Leptin acts on neurons in the hypothalamus and elsewhere to elicit its effects, and mutations that affect the function of this neural circuit cause Mendelian forms of obesity. Leptin levels fall during starvation and elicit adaptive responses in many other physiologic systems, the net effect of which is to reduce energy expenditure. These effects include cessation of menstruation, insulin resistance, alterations of immune function, and neuroendocrine dysfunction, among others. Some or all of these effects are also seen in patients with constitutively low leptin levels, such as occur in lipodystrophy. Leptin is an approved treatment for generalized lipodystrophy, a condition associated with severe metabolic disease, and has also shown potential for the treatment of other types of diabetes. In addition, leptin restores reproductive capacity and increases bone mineral density in patients with hypothalamic amenorrhea, an infertility syndrome in females. Most obese patients have high endogenous levels of leptin, in some instances as a result of mutations in the neural circuit on which leptin acts, though in most cases, the pathogenesis of leptin resistance is not known. Obese patients with leptin resistance show a variable response to exogenous leptin but may respond to a combination of leptin plus amylin. Overall, the identification of leptin has provided a framework for studying the pathogenesis of obesity in the general population, clarified the nature of the biologic response to starvation, and helped to advance our understanding of the neural mechanisms that control feeding.
Journal Article
Drivers and mechanisms of tree mortality in moist tropical forests
Tree mortality rates appear to be increasing in moist tropical forests (MTFs) with significant carbon cycle consequences. Here, we review the state of knowledge regarding MTF tree mortality, create a conceptual framework with testable hypotheses regarding the drivers, mechanisms and interactions that may underlie increasing MTF mortality rates, and identify the next steps for improved understanding and reduced prediction. Increasing mortality rates are associated with rising temperature and vapor pressure deficit, liana abundance, drought, wind events, fire and, possibly, CO2 fertilization-induced increases in stand thinning or acceleration of trees reaching larger, more vulnerable heights. The majority of these mortality drivers may kill trees in part through carbon starvation and hydraulic failure. The relative importance of each driver is unknown. High species diversity may buffer MTFs against large-scale mortality events, but recent and expected trends in mortality drivers give reason for concern regarding increasing mortality within MTFs. Models of tropical tree mortality are advancing the representation of hydraulics, carbon and demography, but require more empirical knowledge regarding the most common drivers and their subsequent mechanisms. We outline critical datasets and model developments required to test hypotheses regarding the underlying causes of increasing MTF mortality rates, and improve prediction of future mortality under climate change.
Journal Article
Correction: Hussain et al. Starvation Protects Hepatocytes from Inflammatory Damage through Paradoxical mTORC1 Signaling. Cells 2023, 12, 1668
In the original publication [...]
Journal Article