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Nadroparin-induced skin necrosis: clinical manifestation of HIT-2 even in the absence of thrombocytopaenia
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Nadroparin-induced skin necrosis: clinical manifestation of HIT-2 even in the absence of thrombocytopaenia
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Nadroparin-induced skin necrosis: clinical manifestation of HIT-2 even in the absence of thrombocytopaenia
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Journal Article
Nadroparin-induced skin necrosis: clinical manifestation of HIT-2 even in the absence of thrombocytopaenia
2016
Overview
Skin necrosis at the site of heparin injections is a well-described complication of treatment with unfractionated or low-molecular weight (LMW) heparin, and should immediately suggest the presence of HIT. The decisive diagnostic procedure in heparin-induced necrosis is HIT antibody testing, histological examination and the chronological connection between the initiation of heparin therapy and the first appearance of skin necrosis; this usually occurs 5–10 days later, either at the site of injection or far from the area of puncture. Learning points Skin necrosis at either the heparin injection site or at a distance, should immediately suggest the presence of heparin-induced thrombocytopaenia (HIT), with a score of 2 points in the 4T's scoring system.
Publisher
BMJ Publishing Group Ltd,BMJ Publishing Group LTD,BMJ Publishing Group
Subject
