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创伤后应激障碍大鼠海马、杏仁核神经元自噬和凋亡改变
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Journal Article
创伤后应激障碍大鼠海马、杏仁核神经元自噬和凋亡改变
2014
Overview
目的观察创伤后应激障碍（PTsD）模型大鼠海马、杏仁核神经元自噬和凋亡的改变，探讨PTSD海马、杏仁核体积异常的机制。方法成年健康雄性SD大鼠40只，随机分为对照组和模型组。采用改良的单一连续应激方法制备PTSD大鼠模型；采用Westernblotting方法检测海马、杏仁核组织LC-3和Beclin1的表达水平；TUNEI，染色和流式细胞术检测海马、杏仁核神经元凋亡情况。结果模型组海马、杏仁核组织LC3Ⅱ／LC3-I比值和Beclin-l表达水平高于对照组（P〈0．05）；模型组海马、杏仁核神经元TUNEI。阳性细胞百分率和凋亡率高于对照组（P〈0．01）。结论PTSD模型大鼠海马、杏仁核神经元存在明显的细胞自噬和凋亡，这可能是海马、杏仁核体积异常的机制之一。
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