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PI3K/AKT通路在非小细胞肺癌顺铂耐药中的作用
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PI3K/AKT通路在非小细胞肺癌顺铂耐药中的作用
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Journal Article
PI3K/AKT通路在非小细胞肺癌顺铂耐药中的作用
2014
Overview
研究表明,PI3K/AKT信号通路在非小细胞肺癌存在异常激活,对肿瘤细胞的增殖、凋亡、存活、耐药等起着重要的作用。顺铂是临床最常用一线化疗药物,但随着治疗的进展肿瘤对其耐药的现象越来越普遍,严重制约其临床效果。顺铂耐药是多种机制共同参与的复杂过程,其中PI3K/AKT通路或其组分持续被激活是非常重要的因素之一。本文对二者关系研究领域的进展作一综述。
