Asset Details
Do you wish to reserve the book?
血红素加氧酶-1与肾脏损伤
Hey, we have placed the reservation for you!
By the way, why not check out events that you can attend while you pick your title.
Oops! Something went wrong.
Looks like we were not able to place the reservation. Kindly try again later.
Are you sure you want to remove the book from the shelf?
血红素加氧酶-1与肾脏损伤
Do you wish to request the book?
血红素加氧酶-1与肾脏损伤
Please be aware that the book you have requested cannot be checked out. If you would like to checkout this book, you can reserve another copy
We have requested the book for you!
Your request is successful and it will be processed during the Library working hours. Please check the status of your request in My Requests.
Oops! Something went wrong.
Looks like we were not able to place your request. Kindly try again later.
Journal Article
血红素加氧酶-1与肾脏损伤
2012
Overview
血红素加氧酶-I（HO-1）作为哺乳动物降解血红素的主要途径之一，其主要降解产物一氧化碳（cO）、自由铁（Fe2＋）和胆绿素--在促进细胞生存、细胞内物质循环及免疫调节中具有重要作用。既往研究提示血红素-HO-l通路是决定急性肾损伤（AKI）易感性及严重性的重要内在因素。诱导Ho-l表达能够减轻肾脏缺血一再灌注损伤（IRI）严重程度，而抑制HO-1的表达会加重IRI。本文综述了国内外有关HO．1在AKI诱导保护机制方面的最新研究进展，以便深入认识HO．1在AKI治疗中的作用。
We currently cannot retrieve any items related to this title. Kindly check back at a later time.