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MATERNAL STRESS, EPIGENETICS, LATERALITY AND THEIR RELATIONSHIP TO FUNCTIONAL DISCONNECTION IN AUTISM
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MATERNAL STRESS, EPIGENETICS, LATERALITY AND THEIR RELATIONSHIP TO FUNCTIONAL DISCONNECTION IN AUTISM
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Journal Article
MATERNAL STRESS, EPIGENETICS, LATERALITY AND THEIR RELATIONSHIP TO FUNCTIONAL DISCONNECTION IN AUTISM
2013
Overview
Both the prevalence and incidence of autism have been confirmed to have increased dramatically over the past two decades. In considering what environmental factors are driving this increase, the researchers must first understand what is actually happening in the autistic brain. They have previously stated and shown that the primary problem is a functional disconnection which is precipitated by a developmental delay of the right hemisphere. Environmental factors along with genetic and epigenetic factors essentially result in either mutating, damaging, deleting or silencing genes that are primarily responsible for the development of functional connections and the growth and maturity of neurons. There have been a number of environmental factors already identified as playing a role in the possible causation of autism including various chemicals, drugs and maternal infection. When looking at the candidate environmental factors that may play a significant role we believe that maternal stress resulting in an elevation of glucocorticoids and inflammation may be the single most significant factor.
Publisher
Nova Science Publishers, Inc
Subject
