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Mitochondrial dysfunction and longevity in animals: Untangling the knot
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Journal Article
Mitochondrial dysfunction and longevity in animals: Untangling the knot
2015
Overview
Mitochondria generate adenosine 5ʹ-triphosphate (ATP) and are a source of potentially toxic reactive oxygen species (ROS). It has been suggested that the gradual mitochondrial dysfunction that is observed to accompany aging could in fact be causal to the aging process. Here we review findings that suggest that age-dependent mitochondrial dysfunction is not sufficient to limit life span. Furthermore, mitochondrial ROS are not always deleterious and can even stimulate pro-longevity pathways. Thus, mitochondrial dysfunction plays a complex role in regulating longevity.
Publisher
American Association for the Advancement of Science,The American Association for the Advancement of Science
