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Proinflammatory IL-17 induces iBALT development
by
Mingzhao Zhu Yangxin Fu
in
Animals
/ Animals, Newborn
/ Antibodies
/ Biomedical and Life Sciences
/ Biomedicine
/ Bronchi - immunology
/ CCL19 protein
/ CCL21 protein
/ Chemokines
/ CXCL13 protein
/ Development
/ Gene Expression Regulation, Developmental - immunology
/ Immunity, Innate
/ Immunology
/ Inflammation
/ Inflammation Mediators - immunology
/ Interleukin 17
/ Interleukin-17 - immunology
/ Interleukin-23 Subunit p19 - immunology
/ Lymphoid Tissue - immunology
/ Lymphotoxin
/ Medical Microbiology
/ Mice
/ Mice, Knockout
/ Microbiology
/ Pneumonia - immunology
/ Receptors, Antigen, T-Cell, gamma-delta - metabolism
/ Research Highlight
/ T-Lymphocyte Subsets - immunology
/ T-Lymphocyte Subsets - metabolism
/ Th17 Cells - immunology
/ Th17 Cells - metabolism
/ Tumor necrosis factor
/ Vaccine
/ 主任
/ 家族
/ 淋巴器官
/ 白细胞介素17
/ 联络
/ 肿瘤坏死因子
/ 诱导
/ 趋化因子
2012
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Proinflammatory IL-17 induces iBALT development
by
Mingzhao Zhu Yangxin Fu
in
Animals
/ Animals, Newborn
/ Antibodies
/ Biomedical and Life Sciences
/ Biomedicine
/ Bronchi - immunology
/ CCL19 protein
/ CCL21 protein
/ Chemokines
/ CXCL13 protein
/ Development
/ Gene Expression Regulation, Developmental - immunology
/ Immunity, Innate
/ Immunology
/ Inflammation
/ Inflammation Mediators - immunology
/ Interleukin 17
/ Interleukin-17 - immunology
/ Interleukin-23 Subunit p19 - immunology
/ Lymphoid Tissue - immunology
/ Lymphotoxin
/ Medical Microbiology
/ Mice
/ Mice, Knockout
/ Microbiology
/ Pneumonia - immunology
/ Receptors, Antigen, T-Cell, gamma-delta - metabolism
/ Research Highlight
/ T-Lymphocyte Subsets - immunology
/ T-Lymphocyte Subsets - metabolism
/ Th17 Cells - immunology
/ Th17 Cells - metabolism
/ Tumor necrosis factor
/ Vaccine
/ 主任
/ 家族
/ 淋巴器官
/ 白细胞介素17
/ 联络
/ 肿瘤坏死因子
/ 诱导
/ 趋化因子
2012
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Proinflammatory IL-17 induces iBALT development
by
Mingzhao Zhu Yangxin Fu
in
Animals
/ Animals, Newborn
/ Antibodies
/ Biomedical and Life Sciences
/ Biomedicine
/ Bronchi - immunology
/ CCL19 protein
/ CCL21 protein
/ Chemokines
/ CXCL13 protein
/ Development
/ Gene Expression Regulation, Developmental - immunology
/ Immunity, Innate
/ Immunology
/ Inflammation
/ Inflammation Mediators - immunology
/ Interleukin 17
/ Interleukin-17 - immunology
/ Interleukin-23 Subunit p19 - immunology
/ Lymphoid Tissue - immunology
/ Lymphotoxin
/ Medical Microbiology
/ Mice
/ Mice, Knockout
/ Microbiology
/ Pneumonia - immunology
/ Receptors, Antigen, T-Cell, gamma-delta - metabolism
/ Research Highlight
/ T-Lymphocyte Subsets - immunology
/ T-Lymphocyte Subsets - metabolism
/ Th17 Cells - immunology
/ Th17 Cells - metabolism
/ Tumor necrosis factor
/ Vaccine
/ 主任
/ 家族
/ 淋巴器官
/ 白细胞介素17
/ 联络
/ 肿瘤坏死因子
/ 诱导
/ 趋化因子
2012
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Journal Article
Proinflammatory IL-17 induces iBALT development
2012
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Overview
The development of secondary lymphoid organs (SLOs) and tertiary lymphoid organs (TLOs) share many common mechanisms. Molecules of the tumor necrosis factor family, especially lymphotoxin, and the lymphoid homeostatic chemokines, such as CXCL13, CCL19 and CCL21, play important roles in the development of both SLOs and TLOs.1-3 This may be unsurprising given the common microarchitectural features of SLOs and TLOs.
Publisher
Nature Publishing Group UK,Nature Publishing Group
Subject
/ Biomedical and Life Sciences
/ Gene Expression Regulation, Developmental - immunology
/ Inflammation Mediators - immunology
/ Interleukin-23 Subunit p19 - immunology
/ Lymphoid Tissue - immunology
/ Mice
/ Receptors, Antigen, T-Cell, gamma-delta - metabolism
/ T-Lymphocyte Subsets - immunology
/ T-Lymphocyte Subsets - metabolism
/ Vaccine
/ 主任
/ 家族
/ 淋巴器官
/ 白细胞介素17
/ 联络
/ 肿瘤坏死因子
/ 诱导
/ 趋化因子
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