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Deficits in perceptual noise exclusion in developmental dyslexia
by
Sperling, Anne J
, Lu, Zhong-Lin
, Manis, Franklin R
, Seidenberg, Mark S
in
Animal Genetics and Genomics
/ Auditory perception in children
/ Behavioral Sciences
/ Biological Techniques
/ Biomedical and Life Sciences
/ Biomedicine
/ brief-communication
/ Dyslexia
/ Dyslexic children
/ Hypotheses
/ Language disorders
/ Neural transmission
/ Neurobiology
/ Neurosciences
/ Phonology
/ Reading comprehension
/ Regulation
2005
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Deficits in perceptual noise exclusion in developmental dyslexia
by
Sperling, Anne J
, Lu, Zhong-Lin
, Manis, Franklin R
, Seidenberg, Mark S
in
Animal Genetics and Genomics
/ Auditory perception in children
/ Behavioral Sciences
/ Biological Techniques
/ Biomedical and Life Sciences
/ Biomedicine
/ brief-communication
/ Dyslexia
/ Dyslexic children
/ Hypotheses
/ Language disorders
/ Neural transmission
/ Neurobiology
/ Neurosciences
/ Phonology
/ Reading comprehension
/ Regulation
2005
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Do you wish to request the book?
Deficits in perceptual noise exclusion in developmental dyslexia
by
Sperling, Anne J
, Lu, Zhong-Lin
, Manis, Franklin R
, Seidenberg, Mark S
in
Animal Genetics and Genomics
/ Auditory perception in children
/ Behavioral Sciences
/ Biological Techniques
/ Biomedical and Life Sciences
/ Biomedicine
/ brief-communication
/ Dyslexia
/ Dyslexic children
/ Hypotheses
/ Language disorders
/ Neural transmission
/ Neurobiology
/ Neurosciences
/ Phonology
/ Reading comprehension
/ Regulation
2005
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Deficits in perceptual noise exclusion in developmental dyslexia
Journal Article
Deficits in perceptual noise exclusion in developmental dyslexia
2005
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Overview
We evaluated signal-noise discrimination in children with and without dyslexia, using magnocellular and parvocellular visual stimuli presented either with or without high noise. Dyslexic children had elevated contrast thresholds when stimuli of either type were presented in high noise, but performed as well as non-dyslexic children when either type was displayed without noise. Our findings suggest that deficits in noise exclusion, not magnocellular processing, contribute to the etiology of dyslexia.
Publisher
Nature Publishing Group US,Nature Publishing Group
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