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No acceleration of recovery from exercise-induced muscle damage after cold or hot water immersion in women: A randomised controlled trial
No acceleration of recovery from exercise-induced muscle damage after cold or hot water immersion in women: A randomised controlled trial
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No acceleration of recovery from exercise-induced muscle damage after cold or hot water immersion in women: A randomised controlled trial
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No acceleration of recovery from exercise-induced muscle damage after cold or hot water immersion in women: A randomised controlled trial
No acceleration of recovery from exercise-induced muscle damage after cold or hot water immersion in women: A randomised controlled trial

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No acceleration of recovery from exercise-induced muscle damage after cold or hot water immersion in women: A randomised controlled trial
No acceleration of recovery from exercise-induced muscle damage after cold or hot water immersion in women: A randomised controlled trial
Journal Article

No acceleration of recovery from exercise-induced muscle damage after cold or hot water immersion in women: A randomised controlled trial

2025
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Overview
This study compared the effects of cold water immersion (CWI) and hot water immersion (HWI) on muscle recovery following a muscle-damaging exercise protocol in women. Thirty healthy women (23.3 ± 2.9 years) were randomly assigned to either the CWI, HWI, or control (CON) groups. Participants completed a standardised exercise protocol (5 x 20 drop-jumps), followed by a 10 min recovery intervention (CWI, HWI, or CON) immediately and 120 min post-exercise. Physiological responses, including muscle oxygen saturation (SmO 2 ), core and skin temperature, and heart rate, were assessed at baseline, immediately post-exercise, after the first recovery intervention (postInt), and during 30 min follow-up. Recovery was evaluated through maximal voluntary isometric contraction, muscle swelling, muscle soreness ratings, and serum creatine kinase at baseline, 24, 48, and 72 h post-exercise. A mixed-effects model was used to account for repeated measures over time. Results showed lower SmO 2 values in the CWI compared to the HWI group at 20 min (Δ-6.76%, CI: −0.27 to −13.25, p = 0.038) and 30 min (Δ-9.86%, CI: −3.37 to −16.35, p = 0.001), and compared to CON at 30 min (Δ-7.28%, CI: −13.77 to −0.79, p = 0.022). Core temperature was significantly higher in the HWI than the CWI group (postInt and 30 min), while it was significantly lower in the CWI group than CON (30 min). CWI caused a substantial decrease in skin temperature compared to HWI and CON between postInt and 30 min follow-up (all p < 0.001). Skin temperature was higher in the HWI group compared to CON at postInt and throughout 30 min follow-up (all p < 0.001). No significant differences in recovery markers were observed between CWI and HWI groups, although HWI led to slightly higher creatine kinase levels (24 h and 72 h) and greater muscle swelling (24 h) compared to CON. Despite distinct acute physiological responses to CWI and HWI, neither improved subjective or objective recovery outcomes during the 72 h follow-up compared to CON in women following a muscle-damaging exercise protocol.